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Causes. It has been suggested that idiopathic nonspecific interstitial pneumonia has an autoimmune mechanism, and is a possible complication of undifferentiated connective tissue disease; [2] however, not enough research has been done at this time to find a cause. Patients with NSIP will often have other unrelated lung diseases like COPD or ...
Desquamative interstitial pneumonia (DIP) is a form of idiopathic interstitial pneumonia featuring elevated numbers of macrophages within the alveoli (air sacs) of the lung. The alveolar macrophages have a characteristic light brown pigmentation and accumulate in the alveolar lumen and septa regions of the lower lobes of the lungs. [1]
Smoking-related interstitial fibrosis (SRIF) is an abnormality in the lungs characterized by excessive collagen deposition within the walls of the air sacs ( interstitial fibrosis ). This abnormality can be seen with a microscope and diagnosed by pathologists. It is caused by cigarette smoking.
Interstitial lung disease ( ILD ), or diffuse parenchymal lung disease ( DPLD ), [3] is a group of respiratory diseases affecting the interstitium (the tissue) and space around the alveoli (air sacs) of the lungs. [4] It concerns alveolar epithelium, pulmonary capillary endothelium, basement membrane, and perivascular and perilymphatic tissues.
Respirology. Idiopathic interstitial pneumonia ( IIP ), or noninfectious pneumonia [1] are a class of diffuse lung diseases. These diseases typically affect the pulmonary interstitium, although some also have a component affecting the airways (for instance, cryptogenic organizing pneumonitis ). There are seven recognized distinct subtypes of IIP.
Acute interstitial pneumonitis (also known as acute interstitial pneumonia) is a rare, severe lung disease that usually affects otherwise healthy individuals. There is no known cause or cure. Acute interstitial pneumonitis is often categorized as both an interstitial lung disease and a form of acute respiratory distress syndrome (ARDS).
Smoker's macrophages are alveolar macrophages whose characteristics, including appearance, cellularity, phenotypes, immune response, and other functions, have been affected upon the exposure to cigarettes. [1] These altered immune cells are derived from several signaling pathways and are able to induce numerous respiratory diseases.
Specialty. Pulmonology. Combined pulmonary fibrosis and emphysema ( CPFE ), describes a medical syndrome involving both pulmonary fibrosis and emphysema. [1] [2] The combination is most commonly found in male smokers. Pulmonary function tests typically show preserved lung volume with very low transfer factor. [3]